ADH Hormone - Be Healthy!

Syndrome of ADH hormone inappropriate antipathetic hormone (DAD) secretion (STAID) defined by hypothermia and hypo resulting commonality of the secretion or action of the hormone continued despite the volume of the normal or inappropriate plasma increased this results in impaired water excretion. The key to understanding the pathologically, signs, symptoms and treatment of STAID is the realization that ADH hormone hypothermia is the result of an excess of water instead of a sodium deficiency. Essential Updated: Patient response to contaminant in neurological intensive care with STAID.

A retrospective study of 32 neurological patients with hypothermia STAID anatomic examined the response to a single bolus of 20 MGM of ADH hormone contaminant for a period of 48 hours. In a base of 129.8 ± 3.4 meg / L, the serum sodium concentration increased to 133.1 ± 3.2 meg / L 6 hours after administration of contaminant and 134.2 ± 3.2 meg / L in 24 hours, indicating a significant response anatomic 24 hours . Patients who did not meet the primary endpoint (increased serum sodium ≥ 4 meg / L during the first 24 hours ADH hormone) were treated with additional doses of contaminant and other agents. Based on these results, the authors recommend a single dose of 20 MGM of contaminant that the focus of initial treatment of choice for patients in the intensive care unit of neurosurgery STAID, the response anatomic 24 hours dictate whether doses are needed contaminant additional or other interventions ADH hormone.

Signs and symptoms. Depending on the scale and pace of development of hypothermia may or may not cause symptoms. The history should take into account the following considerations: In general, slowly progressive hypothermia is associated with fewer symptoms that a rapid decrease in serum sodium occurs at the same value ADH hormone. Signs and symptoms of acute hypothermia does not correspond exactly with the severity or acuity of hypothermia. Patients may have symptoms that suggest an increase in the secretion of DAD, such as chronic pain, symptoms of central nervous system tumors or lung or head injury or drug use.

ADH hormone sources of excessive fluid intake should be evaluated. The chronic of the disease should be considered. After identification of hypothermia, the approach to the patient depends on the volume status clinically evaluated. Important physical signs may be observed in severe hypothermia or rapid onset and may include the following: Confusion, disorientation, delirium. Generalized muscle weakness, anonymous, tremor, asterisks, ADH hormone hyperplasia, ataxia, disarrays, Cheyenne Stokes, pathological reflexes. Generalized seizures, coma.